[Nature] Helicobacter pylori CagA interacts with E-cadherin and deregulates the 汕-catenin signal that promotes intestinal transdifferentiation in gastric epithelial cells |
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Reward10points
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Intestinal cGAS-STING-IFN signalling promotes obesity by downregulating microbiota-derived IAA in male mice
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Two non-active site residues W165 and L166 prominently influence the beta-lactam hydrolytic ability of OXA-23 beta-lactamase
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Helicobacter pylori CagA interacts with E-cadherin and deregulates the 汕-catenin signal that promotes intestinal transdifferentiation in gastric epithelial cells
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Battery integration as function reassignment
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Day每night shifts in microbial activity affecting arsenic and iron in the rice rhizosphere
